Virsuses and cells both contain genetic material and proteins. However, virsuses are smaller than cells and contain no ribosomes or organells. Moreover, virsues do not have a metabolism and they cannot replicate on their own; they must infect a living host cell to replicate.
A virsus's host range is determined by the presence of a specific receptor on the host cell.
Viruses evolve by natural selection. Mutations in viral DNA or RNA create genetic variation; some variants are more sccessful than others at infecting cells and leaving descendants.
The source of energy and raw materials is the host cell's ATP and its stores of nucleotides and amino acids.
1 - Attachment: Virus adheres to host cell receptor.
2 - Penetraiton: Virus enters the cell.
3 - Synthesis: Multiple copies of the viral genome and proteins are produced by the host cell.
4 - Assembly: The viral genetic information is packaged in a protein coat.
5 - Release: New viruses leave host cell.
In a lyric infection, the bacteriophage immediately replicates after infecting a bacterium and then bursts out of the host cell, killing in.
A lysogenic infection is similar to a lytic infection in that they both begin with the attachment and penetration steps. In a lytic infection, the remaining steps (synthesis, assembly, and release) occur immediately, destroying the host bacterium. In contrast, a lysogenic bacteriophage inserts its DNA into the host chromosome, where it is carried on into daughter cells. Eventally an environmental change triggers a switch to the lytic pathway, destroying the infected host cell.
Viral infections can be acquired by inhaled repiratory droplets or ingesting contaminated food and water. Some viruses are also transmitted by blood trasfusion, sexual contact or contaminated needles.
The influenza virus kills cells in the repiratory tact, casing cough and sore throat. The immune system attacks the virus with fever and inflammation, which causes body aches.
After attachment and penetration, the HIV enzyme reverse transcriptase copies the viral RNA to DNA. The viral DNA then insterst itself into the host cell's DNA. New HIV particles are synthesized and assembled inside the host cell and are released by budding. A new virus acquires its envelope from the host cell's membrane as it emerges.
Viral infections are difficult to treat, in part because viruses infect living host cells; it is difficult to destroy the virus without also destroying the host cell. A few antiviral drugs do prevent viral replication, but viruses are genetically variable, and new treatments quickly select for resistant varieties. The best weapon for prevention is a vaccine, which builds immunity to a virus before a person is exposed.
Viruses often enter plant cells by hitching a ride on the contaminated mouthparts of plant-feeding insects. The viruses spread within the plant via plasmodesmata or in vascular tissue.
Some symptoms include small dead spots, mottled leaves, abnormal growth, or even streaking in some flowers.
All viruses have genetic information and a protein coat. Viroids consist only of RNA and therefore lack a protein coat. Prions are abnormally shaped proteins wiht no genetic information.
Viroids interfere with the production of important proteins. An abnormal prion protein "recruits" normal proteins to refold into the abnormal configuration, triggering a chain reaction that results in cell death.
The best way to avoid prion disease is to keep animal brains and spinal cords out of the human food chain and out of medical products.
The team gathered ape droppings from several sites in Cameroon. They tested each sample for SIV antibodies and extracted genetic material from the sample. The genetic information revealed the ape species that produced the feces and the type of SIV virus (if any) that infected the ape.
Virus
Envelope
Bacteriophages
A living cell
Viruses are intracellular parasites that most biologists do not consider to be alive.
The type of genetic material, the shape of the capsi, the presence or absence of an envelope, the replication strategy and the type of disease.
The viral proteins and nucleic acids, then assembles these components into new viruses.
Attachment, Penetration, Synthesis, Assembly, and Release.
Lytic Infection
Lysogenic Infection
Virulence
Latent Infections
killing the infected cells and by stimulating imune responses.
Plasmodesmata
Viroids
Prion
Cytoplasm
Human T Cell
Evolution
The Envelope
A Latent infection in animals
Reverse transcriptase from the virus
Both A and C
A protein that can aalter the shape of a second protein