t helper cell bind to antigen presenting cell and become acitvated. once activatd, they rlease interleukins which stimulate the acivatiom of the t killer cells.
supress the immune responce so that cell mediwted rsponce only occurs when pathogen is acivtated
release oroteins, porforins to crewte hooes and enable cell desth
allows flexibility so the antibody can bind to many pathogens
stores large amount of data
eg dna and amino acid sequences
protein structyre
needed for analysis of large amounts of data
eg preediction of amino acid sewunce
increase permeability of the bacterial cell membrane making it easier for the plasmis containjng the bacteria to enter the bacterial cell
t helper cell bunds to apc
th cell release interleukins
stimulaarw divide by mitosis to create t memory and killer cells
peptide horomone causing anterior pituituwry glamd to release ACTH causimg the adrenal cortex to release cortisol
blood pressure
increases the rate at which is generwtes excitwtions
causes a confonical change in the shape of the troponin- tropomyosin co,plex
involved in muscular movement and coordination